Browsing by Author Shemon, Anne

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Issue DateTitleAuthor(s)Citation
2004An Arg307 To Gln Polymorphism Within The Atp-Binding Site Causes Loss Of Function Of The Human P2X7 Receptor.Barden, Julian; Dao-Ung, L; Fuller, Stephen; Gu, Baijun (Ben); Shemon, Anne; Skarratt, K; Sluyter, Ronald; Wiley, James; Clarke, Alison L; Petrou, Steven; School of Medical Sciences: Anatomy & Histology; Nepean Clinical School: Medicine; Northern Clinical School: Medicine; Nepean Clinical School: Medicine; Nepean Clinical School: Medicine; Nepean Clinical School: Medicine; School of Medical Sciences: Pathology; Nepean Clinical School: MedicineAn Arg307 To Gln Polymorphism Within The Atp-Binding Site Causes Loss Of Function Of The Human P2X7 Receptor., Journal of Biological Chemistry, vol.279,(30),2004,pp 31287-31295
2007Canine erythrocytes express the P2X7 receptor: greatly increased function compared to human erythrocytesEslick, Guy; Georgiou, Jennifer; Shemon, Anne; Sluyter, Ronald; Stevenson, Ryan; Taylor, Rosanne; Wiley, James; Hughes, William E; School of Public Health: Public Health; Nepean Clinical School: Medicine; Nepean Clinical School: Medicine; School of Medical Sciences: Pathology; Nepean Clinical School: Medicine; Vet Science Faculty; Nepean Clinical School: MedicineCanine erythrocytes express the P2X7 receptor: greatly increased function compared to human erythrocytes, American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, vol.293,(5),2007,pp R2090-R2098
2004Chelerythrine And Other Benzophenanthridine Alkaloids Block The Human P2X(7) ReceptorConigrave, Arthur; Shemon, Anne; Sluyter, Ronald; Wiley, James; Molecular & Microbial Bioscien; Nepean Clinical School: Medicine; School of Medical Sciences: Pathology; Nepean Clinical School: MedicineChelerythrine And Other Benzophenanthridine Alkaloids Block The Human P2X(7) Receptor, British Journal of Pharmacology, vol.142,(6),2004,pp 1015-1019
2004Extracellular ATP Increases Cation Fluxes In Human Erythrocytes By Activation Of The P2X7 ReceptorBarden, Julian; Shemon, Anne; Sluyter, Ronald; Wiley, James; School of Medical Sciences: Anatomy & Histology; Nepean Clinical School: Medicine; School of Medical Sciences: Pathology; Nepean Clinical School: MedicineExtracellular ATP Increases Cation Fluxes In Human Erythrocytes By Activation Of The P2X7 Receptor, Journal of Biological Chemistry, vol.279,(43),2004,pp 44749-44755
2004Glu496 To Ala Polymorphism In The P2X7 Receptor Impairs ATP-Induced IL-1 Beta Release From Human Monocytes.Shemon, Anne; Sluyter, Ronald; Wiley, James; Nepean Clinical School: Medicine; School of Medical Sciences: Pathology; Nepean Clinical School: MedicineGlu496 To Ala Polymorphism In The P2X7 Receptor Impairs ATP-Induced IL-1 Beta Release From Human Monocytes., Journal of Immunology, vol.172,(6),2004,pp 3399-3405
2003An Ile-568 to Asn polymorphism prevents normal trafficking and function of the human P2X7 receptorBarden, Julian; Dao-Ung, L; Fuller, Stephen; Gu, Baijun (Ben); Li, Changping; Shemon, Anne; Sluyter, Ronald; Wiley, James; Petrou, Steven; Smart, Megan L; School of Medical Sciences: Anatomy & Histology; Nepean Clinical School: Medicine; Northern Clinical School: Medicine; Nepean Clinical School: Medicine; Nepean Clinical School: Medicine; Nepean Clinical School: Medicine; School of Medical Sciences: Pathology; Nepean Clinical School: MedicineAn Ile-568 to Asn polymorphism prevents normal trafficking and function of the human P2X7 receptor, Journal of Biological Chemistry, vol.278,(19),2003,pp 17108-17113
2007Inhibition of the human P2X7 receptor by a novel protein tyrosine kinase antagonistShemon, Anne; Sluyter, Ronald; Stokes, Leanne; Wiley, James; Manley, Paul W; Nepean Clinical School: Medicine; School of Medical Sciences: Pathology; Nepean Clinical School: Medicine; Nepean Clinical School: MedicineInhibition of the human P2X7 receptor by a novel protein tyrosine kinase antagonist, Biochemical and Biophysical Research Communications (2005), vol.365,(Available online 20 November 2007),2007,pp 515-520
2005Novel protein kinase C Isoforms mediate both extracellular ATP and PMA-induced phospholipase D activity in B-lymphocytesShemon, Anne; Sluyter, Ronald; Wiley, James; Nepean Clinical School: Medicine; School of Medical Sciences: Pathology; Nepean Clinical School: MedicineNovel protein kinase C Isoforms mediate both extracellular ATP and PMA-induced phospholipase D activity in B-lymphocytes, Tissue Antigens, vol.66,(5),2005,pp 541-541
2007P2X(7) receptor activation causes phosphatidylserine exposure in human erythrocytesShemon, Anne; Sluyter, Ronald; Wiley, James; Nepean Clinical School: Medicine; School of Medical Sciences: Pathology; Nepean Clinical School: MedicineP2X(7) receptor activation causes phosphatidylserine exposure in human erythrocytes, Biochemical and Biophysical Research Communications (2005), vol.355,(1),2007,pp 169-173
2006Rottlerin inhibits P2X(7) receptor-stimulated phospholipase D activity in chronic lymphocytic leukaemia B-lymphocytes.Shemon, Anne; Sluyter, Ronald; Wiley, James; Nepean Clinical School: Medicine; School of Medical Sciences: Pathology; Nepean Clinical School: MedicineRottlerin inhibits P2X(7) receptor-stimulated phospholipase D activity in chronic lymphocytic leukaemia B-lymphocytes., Immunology and Cell Biology, vol.85,(1),2006,pp 68-72
2006A Thr(357) to Ser polymorphism in homozygous and compound heterozygous subjects causes absent or reduced P2X(7) function and impairs ATP-induced mycobacterial killing by macrophages.Britton, Warwick; Dao-Ung, L; Fernando, Suran; Fuller, Stephen; Gu, Baijun (Ben); Saunders, Bernadette; Shemon, Anne; Skarratt, K; Sluyter, Ronald; Wiley, James; Clarke, Alison L; Petrou, Steven; Tan, Khai See; Central Clinical School: Centenary Institute; Nepean Clinical School: Medicine; Central Clinical School: Medicine; Northern Clinical School: Medicine; Nepean Clinical School: Medicine; Central Clinical School: Centenary Institute; Nepean Clinical School: Medicine; Nepean Clinical School: Medicine; School of Medical Sciences: Pathology; Nepean Clinical School: MedicineA Thr(357) to Ser polymorphism in homozygous and compound heterozygous subjects causes absent or reduced P2X(7) function and impairs ATP-induced mycobacterial killing by macrophages., Journal of Biological Chemistry, vol.281,(4),2006,pp 2079-2086