Browsing by Author Lai, Fritz

Jump to: 0-9 A B C D E F G H I J K L M N O P Q R S T U V W X Y Z
or enter first few letters:  
Showing results 1 to 6 of 6
Issue DateTitleAuthor(s)Citation
2013The BH3-mimetic ABT-737 sensitizes human melanoma cells to apoptosis induced by selective BRAF inhibitors but does not reverse acquired resistanceGallagher, Stuart; Haass, Nikolas; Hersey, Peter; Mijatov, Branka; Lai, Fritz; Mohana-Kumara, Nethia; Wroblewski, David; Zhang, Xu Dong; Western Clinical School: Medicine (Westmead); Central Clinical School: Centenary Institute; Northern Clinical School: Surgery; Western Clinical School: Westmead Millennium InstituteThe BH3-mimetic ABT-737 sensitizes human melanoma cells to apoptosis induced by selective BRAF inhibitors but does not reverse acquired resistance, Carcinogenesis, vol.34, 2, 2013,pp 237-247
2012Evidence for upregulation of Bim and the splicing factor SRp55 in melanoma cells from patients treated with selective BRAF inhibitorsHersey, Peter; Jiang, Chen Chen; Lai, Fritz; Zhang, Xu Dong; Farrelly, Margaret L.; Northern Clinical School: Surgery; Northern Clinical School: Kolling Institute; Northern Clinical School: Kolling Institute; Northern Clinical School: Kolling InstituteEvidence for upregulation of Bim and the splicing factor SRp55 in melanoma cells from patients treated with selective BRAF inhibitors, Melanoma Research: a journal for basic, translational and clinical research in melanoma, vol.22, 3, 2012,pp 244-251
2012Histone Deacetylases (HDACs) as Mediators of Resistance to Apoptosis in Melanoma and as Targets for Combination Therapy with Selective BRAF InhibitorsGallagher, Stuart; Hersey, Peter; Jin, Lei; Mijatov, Branka; Lai, Fritz; Zhang, Xu Dong; Western Clinical School: Medicine (Westmead); Northern Clinical School: Surgery; Northern Clinical School: Kolling Institute; Western Clinical School: Westmead Millennium InstituteHistone Deacetylases (HDACs) as Mediators of Resistance to Apoptosis in Melanoma and as Targets for Combination Therapy with Selective BRAF Inhibitors, Advances in Pharmacology, vol.65, N/A, 2012,pp 27-43
2014Noxa upregulation by oncogenic activation of MEK/ERK through CREB promotes autophagy in human melanoma cellsScolyer, Richard; Wilmott, James; Guo, Su T; Jiang, Chen Chen; Jin, Lei; Lai, Fritz; Liu, Xiao Ying; Liu, Yi Lun; Luan, Qi; Tseng, Hsin-Yi; Yan, Xu Guang; Zhang, Xu Dong; Central Clinical School: Pathology; Central Clinical School: SurgeryNoxa upregulation by oncogenic activation of MEK/ERK through CREB promotes autophagy in human melanoma cells, Oncotarget, vol.5, 22, 2014,pp 11237-11251
2014Oncogenic Activation of MEK/ERK Primes Melanoma Cells for Adaptation to Endoplasmic Reticulum StressBoyd, Suzanah; Hersey, Peter; Rizos, Helen; Croft, Amanda; Guo, Su T; Jiang, Chen Chen; Jin, Lei; Lai, Fritz; Tay, Kwang Hong; Tseng, Hsin-Yi; Zhang, Xu Dong; Western Clinical School: Medicine (Westmead); Northern Clinical School: Surgery; Western Clinical School: Westmead Millennium InstituteOncogenic Activation of MEK/ERK Primes Melanoma Cells for Adaptation to Endoplasmic Reticulum Stress, Journal of Investigative Dermatology, vol.134, 2, 2014,pp 488-497
2015RIP1 Kinase Is an Oncogenic Driver in MelanomaScolyer, Richard; Wilmott, James; Croft, Amanda; Guo, Sutang; Jiang, Chen Chen; Jin, Lei; Lai, Fritz; Liu, Xiao Ying; Tseng, Hsin-Yi; Wang, ChunYan; Yan, Xu Guang; Zhang, Xu Dong; Central Clinical School: Pathology; Central Clinical School: SurgeryRIP1 Kinase Is an Oncogenic Driver in Melanoma, Cancer Research, vol.75, 8, 2015,pp 1736-1748