Browsing by Author Becker, Therese

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Issue DateTitleAuthor(s)Citation
2012Absence of Distinguishing Senescence Traits in Human Melanocytic NeviBecker, Therese; Gowrishankar, Kavitha; Kefford, Richard (Rick); Rizos, Helen; Scolyer, Richard; Scurr, Lyndee; Thompson, John; Tran, Sieu; Desilva, Chitra; Haferkamp, Sebastian; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Medicine (Westmead); Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium Institute; Central Clinical School: Pathology; Western Clinical School: Medicine (Westmead); Central Clinical School: Surgery; Western Clinical School: Medicine (Westmead)Absence of Distinguishing Senescence Traits in Human Melanocytic Nevi, The Journal of Investigative Dermatology, vol.132, 9, 2012,pp 2226-2234
2012Acquired Resistance to BRAF Inhibition Can Confer Cross-Resistance to Combined BRAF/MEK InhibitionBecker, Therese; Gowrishankar, Kavitha; Kefford, Richard (Rick); Pupo, Gulietta; Rizos, Helen; Snoyman, Stephanie; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Medicine (Westmead); Western Clinical School: Westmead Millennium Institute; Western Clinical School: Medicine (Westmead); Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium InstituteAcquired Resistance to BRAF Inhibition Can Confer Cross-Resistance to Combined BRAF/MEK Inhibition, The Journal of Investigative Dermatology, vol.132, 7, 2012,pp 1850-1859
2010Amino terminal hydrophobic import signals target the p14(ARF) tumor suppressor to the mitochondriaBecker, Therese; Frausto, Monika; Fung, Carina; Irvine, Malama; Kefford, Richard (Rick); Mijatov, Branka; Philipsz, Suzanah; Rizos, Helen; Gallagher, S. J.; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium Institute; Infectious Diseases; Western Clinical School: Medicine (Westmead); Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Medicine (Westmead); Western Clinical School: Westmead Millennium InstituteAmino terminal hydrophobic import signals target the p14(ARF) tumor suppressor to the mitochondria, Cell Cycle, vol.9, 4,pp 829-839
2013Antiproliferative Effects of Continued Mitogen-Activated Protein Kinase Pathway Inhibition Following Acquired Resistance to BRAF and/or MEK Inhibition in MelanomaBecker, Therese; Carlino, Matteo; Gowrishankar, Kavitha; Kefford, Richard (Rick); Long, Georgina; Pupo, Gulietta; Rizos, Helen; Saw, Robyn; Snoyman, Stephanie; Saunders, Catherine; Zhang, Xu Dong; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Medicine (Westmead); Western Clinical School: Medicine (Westmead); Western Clinical School: Westmead Millennium Institute; Central Clinical School: The Sydney Cancer Centre; Western Clinical School: Medicine (Westmead); Western Clinical School: Westmead Millennium Institute; Central Clinical School: Surgery; Western Clinical School: Westmead Millennium InstituteAntiproliferative Effects of Continued Mitogen-Activated Protein Kinase Pathway Inhibition Following Acquired Resistance to BRAF and/or MEK Inhibition in Melanoma, Molecular Cancer Therapeutics, vol.12, 7, 2013,pp 1332-1342
2010Apoptosis of Human Melanoma Cells Induced by Inhibition of B-RAFV600E Involves Preferential Splicing of BimSBecker, Therese; Rizos, Helen; Croft, A.; Hersey, Peter; Jiang, Chen Chen; Lai, F; Liu, H.; Tay, KH; Thorne, R F; Yang, F.; Zhang, Xu Dong; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium InstituteApoptosis of Human Melanoma Cells Induced by Inhibition of B-RAFV600E Involves Preferential Splicing of BimS, Cell Death & Disease, vol.1, 9,pp e69-1-e69-14
2003Association of p14ARF with the p120E4F transciptional represssor enhances cell cycle inhibitionBadhwar, Prerna; Becker, Therese; Diefenbach, Eve; Kefford, Richard (Rick); Rizos, Helen; Woodruff, Sarah; Rooney, Robert J.; Medicine; Western Clinical School: Westmead Millennium Institute; Business Liaison Office; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Medicine (Westmead)Association of p14ARF with the p120E4F transciptional represssor enhances cell cycle inhibition, Journal of Biological Chemistry, vol.278,(7),2003,pp 4981-4989
2002Association of p14ARF with the p120E4F Transcriptional Repressor Enhances Cell Cycle InhibitionBadhwar, Prerna; Becker, Therese; Kefford, Richard (Rick); Rizos, Helen; Diefenbach, Eve; Rooney, Robert J.; Woodruff, Sarah; Medicine; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium InstituteAssociation of p14ARF with the p120E4F Transcriptional Repressor Enhances Cell Cycle Inhibition, Journal of Biological Chemistry, vol.278,(7),2002,pp 4981-4989
2014BRAF inhibitor resistance mechanisms in metastatic melanoma: spectrum and clinical impactBecker, Therese; Boyd, Suzanah; Carlino, Matteo; Fung, Carina; Haydu, Lauren; Howle, Julie; Kefford, Richard (Rick); Long, Georgina; Mijatov, Branka; Pupo, Gulietta; Rizos, Helen; Saw, Robyn; Scolyer, Richard; Thompson, John; Hyman, Jessica; Menzies, Alexander; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Medicine (Westmead); Western Clinical School: Medicine (Westmead); Infectious Diseases; Central Clinical School: Surgery; Western Clinical School: Surgery; Western Clinical School: Westmead Millennium Institute; Central Clinical School: The Sydney Cancer Centre; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Medicine (Westmead); Western Clinical School: Westmead Millennium Institute; Central Clinical School: Surgery; Central Clinical School: Pathology; Central Clinical School: SurgeryBRAF inhibitor resistance mechanisms in metastatic melanoma: spectrum and clinical impact, Clinical Cancer Research, vol.20, 7, 2014,pp 1965-1977
2009The chromatin remodelling factor BRG1 is a novel binding partner of the tumor suppressor p16INK4aBecker, Therese; Diefenbach, Eve; Dijkstra, Menno; Frausto, Monika; Haferkamp, Sebastian; Kefford, Richard (Rick); Mann, Graham; Rizos, Helen; Scolyer, Richard; Scurr, Lyndee; Reisman, David N.; Western Clinical School: Westmead Millennium Institute; Sydnovate; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium Institute; Western Clinical School (Westmead): Office; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium Institute; Central Clinical School: Pathology; Western Clinical School: Medicine (Westmead)The chromatin remodelling factor BRG1 is a novel binding partner of the tumor suppressor p16INK4a, Molecular Cancer, vol.8,(4),2009,pp 4-1-4-12
2002EMelanoBase: An Online Locus-Specific Variant Database for Familial MelanomaBecker, Therese; Fung, David; Mann, Graham; Bressac de Paillerets, Bridgitte; Hayward, Nicholas; Holland, Elizabeth A.; Melanoma Genetics Consortium, MGC; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Medicine (Westmead); Western Clinical School: Westmead Millennium InstituteEMelanoBase: An Online Locus-Specific Variant Database for Familial Melanoma, Human Mutation, vol.21,(1),2002,pp 2-7
2010IGFBP7 is not required for B-RAF-induced melanocyte senescenceBecker, Therese; Haferkamp, Sebastian; Irvine, Malama; Kefford, Richard (Rick); Mann, Graham; Pupo, Gulietta; Rizos, Helen; Scolyer, Richard; Scurr, Lyndee; Becker, Jurgen; Lai, Ken; Schrama, David; Western Clinical School: Westmead Millennium Institute; Western Clinical School (Westmead): Office; Western Clinical School: Medicine (Westmead); Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Medicine (Westmead); Western Clinical School: Westmead Millennium Institute; Central Clinical School: Pathology; Western Clinical School: Medicine (Westmead)IGFBP7 is not required for B-RAF-induced melanocyte senescence, Cell, vol.141, 4,pp 717-727
2005Impaired inhibition of NF-kappaB activity by melanoma-associated p16INK4a mutations.Becker, Therese; Dela Pena, Aileen; Kefford, Richard (Rick); Mann, Graham; Rizos, Helen; Woodruff, Sarah; Leclercq, Isabelle A.; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Medicine (Westmead); Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Medicine (Westmead)Impaired inhibition of NF-kappaB activity by melanoma-associated p16INK4a mutations., Biochemical and Biophysical Research Communications (2005), vol.332,(3),2005,pp 873-879
2014The MAPK pathway functions as a redundant survival signal that reinforces the PI3K cascade in c-Kit mutant melanomaBecker, Therese; Kefford, Richard (Rick); Rizos, Helen; Scurr, Lyndee; Todd, James; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Medicine (Westmead); Molecular BioscienceThe MAPK pathway functions as a redundant survival signal that reinforces the PI3K cascade in c-Kit mutant melanoma, Oncogene, vol.33, 2, 2014,pp 236-245
2005The melanoma-associated 24 base pair duplication in p16INK4a is functionally impairedAyub, Ana; Becker, Therese; Kefford, Richard (Rick); Mann, Graham; Rizos, Helen; Western Clinical School (Westmead): Office; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium InstituteThe melanoma-associated 24 base pair duplication in p16INK4a is functionally impaired, International Journal of Cancer (2005), vol.117,(4),2005,pp 569-573
2013Molecular Mechanisms of Cellular SenescenceBecker, Therese; Haferkamp, Sebastian; Western Clinical School: Westmead Millennium InstituteMolecular Mechanisms of Cellular Senescence in Senescence and Senescence-related disorders, InTech Open Access Publishers (peer reviewed), 2013, pp. 25-50
2014Mutant B-RAF-Mcl-1 survival signaling depends on the STAT3 transcription factorBecker, Therese; Boyd, Suzanah; Gowrishankar, Kavitha; Kefford, Richard (Rick); Mann, Graham; Mijatov, Branka; Pupo, Gulietta; Rizos, Helen; Snoyman, Stephanie; Scolyer, Richard A; Zhang, XD; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Medicine (Westmead); Western Clinical School: Medicine (Westmead); Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Medicine (Westmead); Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium InstituteMutant B-RAF-Mcl-1 survival signaling depends on the STAT3 transcription factor, Oncogene, vol.33, 9, 2014,pp 1158-1166
2009Oncogene-Induced Senescence Does Not Require the p16INK4a or p14ARF Melanoma Tumor SuppressorsBecker, Therese; Frausto, Monika; Haferkamp, Sebastian; Kefford, Richard (Rick); Rizos, Helen; Scurr, Lyndee; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium Institute; Western Clinical School (Westmead): Office; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Medicine (Westmead)Oncogene-Induced Senescence Does Not Require the p16INK4a or p14ARF Melanoma Tumor Suppressors, The Journal of Investigative Dermatology, vol.129, 8, 2009,pp 1983-1991
2013Oncogenic B-RAF(V600E) Signaling Induces the T-Box3 Transcriptional Repressor to Repress E-Cadherin and Enhance Melanoma Cell InvasionBecker, Therese; Boyd, Suzanah; Gowrishankar, Kavitha; Kefford, Richard (Rick); Mann, Graham; Mijatov, Branka; Pupo, Gulietta; Rizos, Helen; Tran, Sieu; Goding, Colin; Scolyer, Richard A; Shaw, Heather M; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Medicine (Westmead); Western Clinical School: Medicine (Westmead); Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Medicine (Westmead); Western Clinical School: Westmead Millennium Institute; Western Clinical School: Medicine (Westmead)Oncogenic B-RAF(V600E) Signaling Induces the T-Box3 Transcriptional Repressor to Repress E-Cadherin and Enhance Melanoma Cell Invasion, The Journal of Investigative Dermatology, vol.133, 5, 2013,pp 1269-1277
2008p16(INK4a)-induced senescence is disabled by melanoma-associated mutationsBecker, Therese; Haferkamp, Sebastian; Kefford, Richard (Rick); Rizos, Helen; Scurr, Lyndee; Western Clinical School: Westmead Millennium Institute; Western Clinical School (Westmead): Office; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Medicine (Westmead)p16(INK4a)-induced senescence is disabled by melanoma-associated mutations, Aging Cell, vol.7, 5, 2008,pp 733-745
2006Physical and functional interaction of the p14ARF tumor suppressor with ribosomes.Ayub, Ana; Becker, Therese; Kefford, Richard (Rick); McKenzie, Heather; Rizos, Helen; Scurr, Lyndee; Woodruff, Sarah; Stahl, Joachim; Western Clinical School (Westmead): Office; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Westmead Millennium Institute; Western Clinical School: Medicine (Westmead); Western Clinical School: Westmead Millennium Institute; Western Clinical School: Medicine (Westmead); Western Clinical School: Medicine (Westmead)Physical and functional interaction of the p14ARF tumor suppressor with ribosomes., Journal of Biological Chemistry, vol.281,(49),2006,pp 38080-38088