Browsing by Author Hahn, Christopher N.

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Issue DateTitleAuthor(s)Citation
2009Chronic increases in sphingosine kinase-1 activity induce a pro-inflammatory, pro-angiogenic phenotype in endothelial cellsGamble, Jennifer; Vadas, Mathew; Xia, Pu; Hahn, Christopher N.; Limaye, Vidya S.; Pitson, Stuart M.; Smith, Malcolm A.; Central Clinical School: Centenary Institute; Central Clinical School: Centenary Institute; Central Clinical School: Centenary InstituteChronic increases in sphingosine kinase-1 activity induce a pro-inflammatory, pro-angiogenic phenotype in endothelial cells, Cellular and Molecular Biology Letters, vol.14, 3, 2009,pp 424-441
2009Sphingosine kinase-1 associates with integrin {alpha}V{beta}3 to mediate endothelial cell survivalGamble, Jennifer; Vadas, Mathew; Bonder, Claudine S; Hahn, Christopher N.; Li, Xiaochun Chun; Pitson, Stuart M.; Sun, Wai Y; Central Clinical School: Centenary Institute; Central Clinical School: Centenary InstituteSphingosine kinase-1 associates with integrin {alpha}V{beta}3 to mediate endothelial cell survival, The American Journal of Pathology: cellular and molecular biology of disease, vol.175, 5,pp 2217-2225
2010Stress-induced premature senescence mediated by a novel gene, SENEX, results in an anti-inflammatory phenotype in endothelial cellsBeck, Konstanze; Gamble, Jennifer; Grimshaw, Matthew; Stocker, Roland; Vadas, Mathew; Brautigan, Peter J.; Coleman, Paul; Hahn, Christopher N.; Li, Xiaochun; Lu, Ying; School of Medical Sciences: School of Medical Sciences; Central Clinical School: Centenary Institute; Central Clinical School: Centenary Institute; School of Medical Sciences: Pathology; Central Clinical School: Centenary InstituteStress-induced premature senescence mediated by a novel gene, SENEX, results in an anti-inflammatory phenotype in endothelial cells, Blood, vol.116, 19,pp 4016-4024